Amarbir Singh Gill, MD

Amarbir Singh Gill, M.D. is an Assistant Professor of Otolaryngology and fellowship trained rhinologist and skull base surgeon. He obtained his medical degree from the George Washington University School of Medicine. Dr. Gill then completed his otolaryngology residency at the University of California Davis before heading to the University of Utah, where he completed a 2-year rhinology and skull base fellowship. He is board certified in otolaryngology.

His clinical interest lies in the diagnosis and management of inflammatory sinus disease, including chronic sinusitis, as well as both sinonasal and skull base tumors, such as pituitary tumors masses, through the use of minimally invasive, endoscopic techniques. He values personalizing care to the needs and wants of each unique patient. Dr. Gill works closely with a multi-disciplinary team of neurosurgeons, head and neck surgeons and neuroradiologists to ensure the best treatment outcomes for his patients. He serves as a faculty member in Head and Neck Surgery.

Acute and chronic sinusitis, nasal polyps and nasal masses, endoscopic surgery, allergic rhinitis, sinus and skull base tumors, cerebrospinal fluid (CSF) leaks

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I am an Assistant Professor of Otolaryngology and surgeon-scientist at the University of Michigan (UM). Both my clinical practice and my research career focus on chronic rhinosinusitis (CRS), an upper airway inflammatory disorder. To this end, I have published over 40 papers on CRS, focusing on disease pathophysiology and mechanisms, environmental exposures, and disease outcomes. I have presented my findings biannually at national conferences during the past 4 years.

My team and I seek to understand the molecular mechanisms driving the development and progression of chronic rhinosinusitis with nasal polyps (CRSwNP), a type 2 inflammatory subtype of CRS. I have expertise in CRSwNP disease phenotyping and endotyping, as well as in elucidation of molecular pathways and targets. My translational research effort combines patient tissue specimen analyses with cell culture and mouse models. My main areas of research focus are: 1) the contribution of macrophages and lipid metabolites to CRSwNP development and disease progression; 2) the differential pathogenic potential of wildfire vs. non-wildfire PM2.5 exposures on CRSwNP; and 3) the development of novel, topical therapeutics strategies for improving CRS outcomes.