We have significant experience treating not only straightforward abdominal aortic aneurysms (AAA), but those that require complex or hybrid techniques.
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Anticoagulants (sometimes known as “blood thinners”) are drugs that prevent blood from clotting or prevent existing clots from getting larger. They can keep harmful clots from forming in the heart, veins or arteries.
The University of Michigan Frankel Cardiovascular Center offers both open heart surgery and minimally invasive treatments for aortic aneurysm.
University of Michigan Frankel Cardiovascular Center Aortic Disease program began in 1995 and has a long history of treating all types of aortic disease.
Endovascular aneurysm repair (EVAR) is a preferred treatment for many abdominal aortic aneurysm patients and an alternative for some who do not qualify for open surgery. Compared with open AAA repair, minimally invasive EVAR is associated with a significant reduction in mortality, primarily because EVAR does not require exposure of the aorta. EVAR also results in a shorter hospital stay and faster recovery time.
The Fenestrated Endograft (FEVAR) is a relatively new minimally-invasive option for people with abdominal aortic aneurysms who don’t qualify for traditional endovascular aneurysm repair (EVAR). The unique feature of fenestrated endografts is that they can cover branch arteries of the aorta (such as the renal arteries) because the graft has fenestrations, or holes, that correspond to the position of the branching arteries within the aorta to allow for blood to flow through the graft into the branch vessel.
The difference between peripheral artery disease or PAD (sometimes called peripheral vascular disease) and heart disease is that the blockages are outside your heart, usually in the legs. The University of Michigan’s Peripheral Arterial Disease Program brings together a multidisciplinary team of physicians to create a treatment plan tailored to your needs.
Pulmonary emboli can present as acute PE or chronic PE. Acute PE is a new obstruction causing acute onset heart strain and often needs immediate treatment with clot busters and blood thinning medications. Chronic PE is a more insidious presentation that includes heart failure with gradual progressive symptoms and is caused by an older residual obstruction resulting from an undissolved clot in the pulmonary circulation left over from previous acute pulmonary emboli. In addition, in a small percentage of patients, chronic PE can lead to elevated blood pressure in the pulmonary arteries over time, developing into a rare type of pulmonary hypertension called chronic thromboembolic pulmonary hypertension (CTEPH).
A thoracic aortic aneurysm is an enlargement in the upper part of the aorta, the major blood vessel that routes blood to the body. Thoracic aortic aneurysms can cause aortic dissection (splitting of the aortic wall) and aortic rupture, leading to life-threatening internal bleeding. Thoracic aortic aneurysms, even those that are large, frequently do not cause symptoms. Some individuals, however, may experience symptoms such as: pain in the chest area; back pain; coughing or hoarseness; and difficulty breathing.